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・ Acute eosinophilic leukemia
・ Acute eosinophilic pneumonia
・ Acute erythroid leukemia
・ Acute esophageal necrosis
・ Acute exacerbation of chronic obstructive pulmonary disease
・ Acute Exposure Guideline Levels
・ Acute fatty liver of pregnancy
・ Acute flaccid myelitis
・ Acute generalized exanthematous pustulosis
・ Acute haemorrhagic conjunctivitis in Ghana
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Acute inhalation injury
・ Acute intermittent porphyria
・ Acute interstitial pneumonitis
・ Acute kidney injury
・ Acute leukemia
・ Acute limb ischaemia
・ Acute liver failure
・ Acute Local Blood Flow Regulation
・ Acute lymphoblastic leukemia
・ Acute medicine
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Acute inhalation injury : ウィキペディア英語版
Acute inhalation injury

Acute inhalation injury may result from frequent and widespread use of household cleaning agents and industrial gases (including chlorine and ammonia). The airways and lungs receive continuous first-pass exposure to non-toxic and irritant or toxic gases via inhalation. Irritant gases are those that, on inhalation, dissolve in the water of the respiratory tract mucosa and provoke an inflammatory response, usually from the release of acidic or alkaline radicals.〔Bessac BF, Jordt SE. (2010) Sensory detection and responses to toxic gases: mechanisms, health effects, and countermeasures. Proc Am Thorac Soc. 7: 269-77.〕〔do Pico GA. (1995) Toxic gas inhalation. Curr Opin Pulm Med. 1:102-8.〕 Smoke, chlorine, phosgene, sulfur dioxide, hydrogen chloride, hydrogen sulfide, nitrogen dioxide, ozone, and ammonia are common irritants.
Depending on the type and amount of irritant gas inhaled, victims can experience symptoms ranging from minor respiratory discomfort to acute airway and lung injury and even death. A common response cascade to a variety of irritant gases includes inflammation, edema and epithelial sloughing, which, left untreated, can result in scar formation and pulmonary and airway remodeling. Currently, mechanical ventilation remains the therapeutic mainstay for pulmonary dysfunction following acute inhalation injury.
== Causes ==


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